You are here
CERID Bibliography
Export 100 results:
Author Title Type [ Year] Filters: Keyword is Antigens, Bacterial [Clear All Filters]
Lipoproteins are major targets of the polyclonal human T cell response to Mycobacterium tuberculosis. J Immunol. 2013 ;190(1):278-84.
. Looking within the zebrafish to understand the tuberculous granuloma. Adv Exp Med Biol. 2013 ;783:251-66.
. Association and evidence for linked recognition of type IV secretion system proteins VirB9-1, VirB9-2, and VirB10 in Anaplasma marginale. Infect Immun. 2012 ;80(1):215-27.
. Expansion of variant diversity associated with a high prevalence of pathogen strain superinfection under conditions of natural transmission. Infect Immun. 2012 ;80(7):2354-60.
. Genome-wide screening and identification of antigens for rickettsial vaccine development. FEMS Immunol Med Microbiol. 2012 ;64(1):115-9.
. . CD1b tetramers bind αβ T cell receptors to identify a mycobacterial glycolipid-reactive T cell repertoire in humans. J Exp Med. 2011 ;208(9):1741-7.
Identification of Anaplasma marginale outer membrane protein antigens conserved between A. marginale sensu stricto strains and the live A. marginale subsp. centrale vaccine. Infect Immun. 2011 ;79(3):1311-8.
. Robust antigen specific th17 T cell response to group A Streptococcus is dependent on IL-6 and intranasal route of infection. PLoS Pathog. 2011 ;7(9):e1002252.
. Antigenic variation in Treponema pallidum: TprK sequence diversity accumulates in response to immune pressure during experimental syphilis. J Immunol. 2010 ;184(7):3822-9.
. The immunization-induced antibody response to the Anaplasma marginale major surface protein 2 and its association with protective immunity. Vaccine. 2010 ;28(21):3741-7.
. Tuberculous granuloma induction via interaction of a bacterial secreted protein with host epithelium. Science. 2010 ;327(5964):466-9.
. Generation of antigenic variants via gene conversion: Evidence for recombination fitness selection at the locus level in Anaplasma marginale. Infect Immun. 2009 ;77(8):3181-7.
. 'Nothing is permanent but change'- antigenic variation in persistent bacterial pathogens. Cell Microbiol. 2009 ;11(12):1697-705.
. High-throughput identification of T-lymphocyte antigens from Anaplasma marginale expressed using in vitro transcription and translation. J Immunol Methods. 2008 ;332(1-2):129-41.
. Rapid deletion of antigen-specific CD4+ T cells following infection represents a strategy of immune evasion and persistence for Anaplasma marginale. J Immunol. 2008 ;181(11):7759-69.
. Superinfection as a driver of genomic diversification in antigenically variant pathogens. Proc Natl Acad Sci U S A. 2008 ;105(6):2123-7.
. Gene conversion is a convergent strategy for pathogen antigenic variation. Trends Parasitol. 2007 ;23(9):408-13.
. Selection for simple major surface protein 2 variants during Anaplasma marginale transmission to immunologically naïve animals. Infect Immun. 2007 ;75(3):1502-6.
. Differential expression and sequence conservation of the Anaplasma marginale msp2 gene superfamily outer membrane proteins. Infect Immun. 2006 ;74(6):3471-9.
. Insights into mechanisms of bacterial antigenic variation derived from the complete genome sequence of Anaplasma marginale. Ann N Y Acad Sci. 2006 ;1078:15-25.
. The CD4+ T cell immunodominant Anaplasma marginale major surface protein 2 stimulates gammadelta T cell clones that express unique T cell receptors. J Leukoc Biol. 2005 ;77(2):199-208.
. CD4+ T cells and toll-like receptors recognize Salmonella antigens expressed in bacterial surface organelles. Infect Immun. 2005 ;73(3):1350-6.
. Complete genome sequencing of Anaplasma marginale reveals that the surface is skewed to two superfamilies of outer membrane proteins. Proc Natl Acad Sci U S A. 2005 ;102(3):844-9.
. .