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CERID Bibliography
Activation of protein kinase C induces rapid internalization and subsequent degradation of muscarinic acetylcholine receptors in neuroblastoma cells. J Biol Chem. 1986 ;261(12):5307-13.
. Alteration in the regulation of neuronal muscarinic acetylcholine receptor number induced by chronic lithium in neuroblastoma cells. Brain Res. 1988 ;439(1-2):88-94.
. Biochemical and immunological studies on the regulation of cardiac and neuronal muscarinic acetylcholine receptor number and function. Trends Pharmacol Sci. 1988 ;Suppl:25-8.
. Cellular FLIP is expressed in cardiomyocytes and down-regulated in TUNEL-positive grafted cardiac tissues. Cardiovasc Res. 2000 ;48(1):101-10.
Clinical implications of mutations of neutrophil elastase in congenital and cyclic neutropenia. J Pediatr Hematol Oncol. 2001 ;23(4):208-10.
. Combined administration of G-CSF and dexamethasone for the mobilization of granulocytes in normal donors: optimization of dosing. Transfusion. 2000 ;40(6):642-4.
. A comparative trial of granulocyte-colony-stimulating factor and dexamethasone, separately and in combination, for the mobilization of neutrophils in the peripheral blood of normal volunteers. Transfusion. 1997 ;37(2):182-7.
. Comparison of interferon-gamma, granulocyte colony-stimulating factor, and granulocyte-macrophage colony-stimulating factor for priming leukocyte-mediated hyphal damage of opportunistic fungal pathogens. J Infect Dis. 1999 ;179(4):1038-41.
. Cross-linking of CD18 primes human neutrophils for activation of the respiratory burst in response to specific stimuli: implications for adhesion-dependent physiological responses in neutrophils. J Leukoc Biol. 1995 ;58(6):690-7.
. Cross-linking of CD45 enhances activation of the respiratory burst in response to specific stimuli in human phagocytes. J Immunol. 1995 ;155(4):2175-84.
. Current status of granulocyte (neutrophil) transfusion therapy for infectious diseases. J Infect Dis. 2001 ;183(2):321-328.
. Decreased muscarinic acetylcholine receptor number in the central nervous system of the tottering (tg/tg) mouse. J Neurochem. 1986 ;46(3):977-82.
. Differential expression of Fas (CD95) and Fas ligand on normal human phagocytes: implications for the regulation of apoptosis in neutrophils. J Exp Med. 1996 ;184(2):429-40.
. Differential induction of apoptosis by Fas-Fas ligand interactions in human monocytes and macrophages. J Exp Med. 1997 ;185(8):1511-6.
. Effects of granulocyte-macrophage colony-stimulating factor (GM-CSF) on neutrophil kinetics and function in normal human volunteers. Am J Hematol. 1998 ;57(1):7-15.
. Emerging role of apoptosis in the pathogenesis of severe neutropenia. Curr Opin Hematol. 2000 ;7(3):131-2.
. Endogenous bacterial endophthalmitis. Report of a ten-year retrospective study. Ophthalmology. 1994 ;101(5):832-8.
. Fas (CD95) induces alveolar epithelial cell apoptosis in vivo: implications for acute pulmonary inflammation. Am J Pathol. 2001 ;158(1):153-61.
. Fas/FADD-mediated activation of a specific program of inflammatory gene expression in vascular smooth muscle cells. Nat Med. 2000 ;6(7):790-6.
. Fas/Fas ligand system mediates epithelial injury, but not pulmonary host defenses, in response to inhaled bacteria. Infect Immun. 2001 ;69(9):5768-76.
. Glucocorticoids inhibit apoptosis of human neutrophils. Blood. 1995 ;86(8):3181-8.
. Granulocyte colony-stimulating factor administered in vivo augments neutrophil-mediated activity against opportunistic fungal pathogens. J Infect Dis. 1997 ;175(4):1012-5.
. Granulocyte transfusion therapy: update on potential clinical applications. Curr Opin Hematol. 2001 ;8(3):161-4.
. Human monocytic cells contain high levels of intracellular Fas ligand: rapid release following cellular activation. J Immunol. 1997 ;159(4):1594-8.
. Immunomodulatory approaches to augment phagocyte-mediated host defense for treatment of infectious diseases. Semin Respir Infect. 2001 ;16(1):11-7.
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