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CERID Bibliography
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Author Title Type [ Year] Filters: Keyword is Lipopolysaccharides [Clear All Filters]
Syndecan-4 regulates early neutrophil migration and pulmonary inflammation in response to lipopolysaccharide. Am J Respir Cell Mol Biol. 2012 ;47(2):196-202.
Differential flux of macrophage inflammatory protein-2 and cytokine-induced neutrophil chemoattractant from the lung after intrapulmonary delivery. Am J Physiol Lung Cell Mol Physiol. 2011 ;301(4):L568-74.
. Effects of age on the synergistic interactions between lipopolysaccharide and mechanical ventilation in mice. Am J Respir Cell Mol Biol. 2010 ;43(4):475-86.
. Mycobacterium tuberculosis lipoprotein LprG (Rv1411c) binds triacylated glycolipid agonists of Toll-like receptor 2. Nat Struct Mol Biol. 2010 ;17(9):1088-95.
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Effect of Toll-like receptor 4 blockade on pulmonary inflammation caused by mechanical ventilation and bacterial endotoxin. Exp Lung Res. 2008 ;34(5):225-43.
. Toll-like receptor stimulation induces higher TNF-alpha secretion in peripheral blood mononuclear cells from patients with hyper IgE syndrome. Int Arch Allergy Immunol. 2008 ;146(3):190-4.
Prevention of LPS-induced acute lung injury in mice by mesenchymal stem cells overexpressing angiopoietin 1. PLoS Med. 2007 ;4(9):e269.
. Transcription factor expression in lipopolysaccharide-activated peripheral-blood-derived mononuclear cells. Proc Natl Acad Sci U S A. 2007 ;104(41):16245-50.
Computational identification of key biological modules and transcription factors in acute lung injury. Am J Respir Crit Care Med. 2006 ;173(6):653-8.
. Dual ligand stimulation of RAW 264.7 cells uncovers feedback mechanisms that regulate TLR-mediated gene expression. J Immunol. 2006 ;177(7):4299-310.
. The Duffy antigen modifies systemic and local tissue chemokine responses following lipopolysaccharide stimulation. J Immunol. 2006 ;177(11):8086-94.
Mechanical ventilation interacts with endotoxemia to induce extrapulmonary organ dysfunction. Crit Care. 2006 ;10(5):R136.
. Hyper-IgE syndrome is not associated with defects in several candidate toll-like receptor pathway genes. Hum Immunol. 2005 ;66(7):842-7.
. Modulation of lipopolysaccharide-induced gene transcription and promotion of lung injury by mechanical ventilation. J Immunol. 2005 ;175(5):3369-76.
. Suppressor alphabeta T lymphocytes control innate resistance to endotoxic shock. J Infect Dis. 2005 ;192(6):1039-46.
. Tissue inhibitor of metalloproteinase-1 deficiency amplifies acute lung injury in bleomycin-exposed mice. Am J Respir Cell Mol Biol. 2005 ;33(3):271-9.
. Enhancement of the endotoxin recognition pathway by ventilation with a large tidal volume in rabbits. Am J Physiol Lung Cell Mol Physiol. 2004 ;286(6):L1114-21.
Mechanical ventilation with moderate tidal volumes synergistically increases lung cytokine response to systemic endotoxin. Am J Physiol Lung Cell Mol Physiol. 2004 ;287(3):L533-42.
. Sustained lipopolysaccharide-induced lung inflammation in mice is attenuated by functional deficiency of the Fas/Fas ligand system. Clin Diagn Lab Immunol. 2004 ;11(2):358-61.
. Pyogenic bacterial infections in humans with IRAK-4 deficiency. Science. 2003 ;299(5615):2076-9.
High and low inflammatory response phenotypes in 101 normal human subjects. Chest. 2002 ;121(3 Suppl):87S-88S.
. Leishmania major activates IL-1 alpha expression in macrophages through a MyD88-dependent pathway. Microbes Infect. 2002 ;4(8):763-71.
. The effect of endotoxin on in vivo rat alveolar macrophage phagocytosis. Exp Lung Res. 1998 ;24(6):745-58.
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