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CERID Bibliography
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Author Title Type [ Year] Filters: Keyword is Adaptor Proteins, Signal Transducing [Clear All Filters]
An attenuating mutation in a neurovirulent Sindbis virus strain interacts with the IPS-1 signaling pathway in vivo. Virology. 2013 ;435(2):269-80.
. IRF-3, IRF-5, and IRF-7 coordinately regulate the type I IFN response in myeloid dendritic cells downstream of MAVS signaling. PLoS Pathog. 2013 ;9(1):e1003118.
Control of innate immune signaling and membrane targeting by the Hepatitis C virus NS3/4A protease are governed by the NS3 helix α0. J Virol. 2012 ;86(6):3112-20.
. Convergent evolution of escape from hepaciviral antagonism in primates. PLoS Biol. 2012 ;10(3):e1001282.
. Thymic stromal lymphopoietin is induced by respiratory syncytial virus-infected airway epithelial cells and promotes a type 2 response to infection. J Allergy Clin Immunol. 2012 ;130(5):1187-1196.e5.
. Mitochondrial-associated endoplasmic reticulum membranes (MAM) form innate immune synapses and are targeted by hepatitis C virus. Proc Natl Acad Sci U S A. 2011 ;108(35):14590-5.
. IPS-1 is essential for the control of West Nile virus infection and immunity. PLoS Pathog. 2010 ;6(2):e1000757.
Induction of IFN-beta and the innate antiviral response in myeloid cells occurs through an IPS-1-dependent signal that does not require IRF-3 and IRF-7. PLoS Pathog. 2009 ;5(10):e1000607.
. Long double-stranded RNA induces an antiviral response independent of IFN regulatory factor 3, IFN-beta promoter stimulator 1, and IFN. J Immunol. 2009 ;183(10):6545-53.
. Distinct RIG-I and MDA5 signaling by RNA viruses in innate immunity. J Virol. 2008 ;82(1):335-45.
Establishment and maintenance of the innate antiviral response to West Nile Virus involves both RIG-I and MDA5 signaling through IPS-1. J Virol. 2008 ;82(2):609-16.
. The innate immune response to Salmonella enterica serovar Typhimurium by macrophages is dependent on TREM2-DAP12. Infect Immun. 2008 ;76(6):2439-47.
. Functional and therapeutic analysis of hepatitis C virus NS3.4A protease control of antiviral immune defense. J Biol Chem. 2007 ;282(14):10792-803.
. . Myeloid differentiation primary response gene (88)- and toll-like receptor 2-deficient mice are susceptible to infection with aerosolized Legionella pneumophila. J Infect Dis. 2006 ;193(12):1693-702.
. Viral and therapeutic control of IFN-beta promoter stimulator 1 during hepatitis C virus infection. Proc Natl Acad Sci U S A. 2006 ;103(15):6001-6.
Hyper-IgE syndrome is not associated with defects in several candidate toll-like receptor pathway genes. Hum Immunol. 2005 ;66(7):842-7.
. Differential role of MyD88 in macrophage-mediated responses to opportunistic fungal pathogens. Infect Immun. 2003 ;71(9):5280-6.
. . Leishmania major activates IL-1 alpha expression in macrophages through a MyD88-dependent pathway. Microbes Infect. 2002 ;4(8):763-71.
. P52rIPK regulates the molecular cochaperone P58IPK to mediate control of the RNA-dependent protein kinase in response to cytoplasmic stress. Biochemistry. 2002 ;41(39):11878-87.
. The innate immune response to bacterial flagellin is mediated by Toll-like receptor 5. Nature. 2001 ;410(6832):1099-103.
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Regulation of interferon-induced protein kinase PKR: modulation of P58IPK inhibitory function by a novel protein, P52rIPK. Mol Cell Biol. 1998 ;18(2):859-71.
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