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CERID Bibliography
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Author Title Type [ Year] Filters: Keyword is Macrophages, Peritoneal [Clear All Filters]
Acyl-CoA synthetase 1 is induced by Gram-negative bacteria and lipopolysaccharide and is required for phospholipid turnover in stimulated macrophages. J Biol Chem. 2013 ;288(14):9957-70.
Innate immune detection of flagellin positively and negatively regulates salmonella infection. PLoS One. 2013 ;8(8):e72047.
. Statins fail to improve outcome in experimental cerebral malaria and potentiate Toll-like receptor-mediated cytokine production by murine macrophages. Am J Trop Med Hyg. 2009 ;81(4):631-7.
. PSGL-1 and mTOR regulate translation of ROCK-1 and physiological functions of macrophages. EMBO J. 2007 ;26(2):505-15.
. Toll-like receptor 4 dependence of innate and adaptive immunity to Salmonella: importance of the Kupffer cell network. J Immunol. 2004 ;172(10):6202-8.
. The p17 cleaved form of caspase-3 is present within viable macrophages in vitro and in atherosclerotic plaque. Arterioscler Thromb Vasc Biol. 2003 ;23(7):1276-82.
. Cu,Zn superoxide dismutase of Mycobacterium tuberculosis contributes to survival in activated macrophages that are generating an oxidative burst. Infect Immun. 2001 ;69(8):4980-7.
. In vitro and in vivo assessment of Salmonella enterica serovar Typhimurium DT104 virulence. Infect Immun. 2001 ;69(7):4673-7.
. Antimicrobial actions of the NADPH phagocyte oxidase and inducible nitric oxide synthase in experimental salmonellosis. I. Effects on microbial killing by activated peritoneal macrophages in vitro. J Exp Med. 2000 ;192(2):227-36.
. Salmonella pathogenicity island 2-dependent evasion of the phagocyte NADPH oxidase. Science. 2000 ;287(5458):1655-8.
. Analysis of virulence of clinical isolates of Salmonella enteritidis in vivo and in vitro. Infect Immun. 1999 ;67(11):5651-7.
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Periplasmic superoxide dismutase protects Salmonella from products of phagocyte NADPH-oxidase and nitric oxide synthase. Proc Natl Acad Sci U S A. 1997 ;94(25):13997-4001.
. A cytolysin encoded by Salmonella is required for survival within macrophages. Proc Natl Acad Sci U S A. 1994 ;91(2):489-93.
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