You are here
CERID Bibliography
Export 28 results:
Author Title Type [ Year] Filters: Keyword is West Nile virus [Clear All Filters]
CD22 is required for protection against West Nile virus Infection. J Virol. 2013 ;87(6):3361-75.
. IRF-3, IRF-5, and IRF-7 coordinately regulate the type I IFN response in myeloid dendritic cells downstream of MAVS signaling. PLoS Pathog. 2013 ;9(1):e1003118.
A systems biology approach reveals that tissue tropism to West Nile virus is regulated by antiviral genes and innate immune cellular processes. PLoS Pathog. 2013 ;9(2):e1003168.
. West Nile virus infection and immunity. Nat Rev Microbiol. 2013 ;11(2):115-28.
. 2'-O methylation of the viral mRNA cap by West Nile virus evades ifit1-dependent and -independent mechanisms of host restriction in vivo. PLoS Pathog. 2012 ;8(5):e1002698.
. Cell-intrinsic innate immune control of West Nile virus infection. Trends Immunol. 2012 ;33(10):522-30.
. IL-1β signaling promotes CNS-intrinsic immune control of West Nile virus infection. PLoS Pathog. 2012 ;8(11):e1003039.
Infectious clones of novel lineage 1 and lineage 2 West Nile virus strains WNV-TX02 and WNV-Madagascar. J Virol. 2012 ;86(14):7704-9.
. The RIG-I-like receptor LGP2 controls CD8(+) T cell survival and fitness. Immunity. 2012 ;37(2):235-48.
Inhibitor of κB kinase epsilon (IKK(epsilon)), STAT1, and IFIT2 proteins define novel innate immune effector pathway against West Nile virus infection. J Biol Chem. 2011 ;286(52):44412-23.
. Interferon regulatory factor-1 (IRF-1) shapes both innate and CD8(+) T cell immune responses against West Nile virus infection. PLoS Pathog. 2011 ;7(9):e1002230.
. Nitric oxide controls an inflammatory-like Ly6C(hi)PDCA1+ DC subset that regulates Th1 immune responses. J Leukoc Biol. 2011 ;89(3):443-55.
. 2'-O methylation of the viral mRNA cap evades host restriction by IFIT family members. Nature. 2010 ;468(7322):452-6.
The innate immune adaptor molecule MyD88 restricts West Nile virus replication and spread in neurons of the central nervous system. J Virol. 2010 ;84(23):12125-38.
. IPS-1 is essential for the control of West Nile virus infection and immunity. PLoS Pathog. 2010 ;6(2):e1000757.
Evasion and disruption of innate immune signalling by hepatitis C and West Nile viruses. Cell Microbiol. 2009 ;11(6):880-8.
. Induction of IFN-beta and the innate antiviral response in myeloid cells occurs through an IPS-1-dependent signal that does not require IRF-3 and IRF-7. PLoS Pathog. 2009 ;5(10):e1000607.
. Measure and countermeasure: type I IFN (IFN-alpha/beta) antiviral response against West Nile virus. J Innate Immun. 2009 ;1(5):435-45.
. Establishment and maintenance of the innate antiviral response to West Nile Virus involves both RIG-I and MDA5 signaling through IPS-1. J Virol. 2008 ;82(2):609-16.
. Interferon regulatory factor IRF-7 induces the antiviral alpha interferon response and protects against lethal West Nile virus infection. J Virol. 2008 ;82(17):8465-75.
. Toll-like receptor 3 has a protective role against West Nile virus infection. J Virol. 2008 ;82(21):10349-58.
. Cell-specific IRF-3 responses protect against West Nile virus infection by interferon-dependent and -independent mechanisms. PLoS Pathog. 2007 ;3(7):e106.
. Innate immune evasion by hepatitis C virus and West Nile virus. Cytokine Growth Factor Rev. 2007 ;18(5-6):535-44.
. PKR and RNase L contribute to protection against lethal West Nile Virus infection by controlling early viral spread in the periphery and replication in neurons. J Virol. 2006 ;80(14):7009-19.
. Resistance to alpha/beta interferon is a determinant of West Nile virus replication fitness and virulence. J Virol. 2006 ;80(19):9424-34.
.