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CERID Bibliography

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2011
Kim H, Higgins S, W Liles C, Kain KC. Endothelial activation and dysregulation in malaria: a potential target for novel therapeutics. Curr Opin Hematol. 2011 ;18(3):177-85.
Lee WL, W Liles C. Endothelial activation, dysfunction and permeability during severe infections. Curr Opin Hematol. 2011 ;18(3):191-6.
R de Claro A, Zhu X, Tang J, Morgan-Stevenson V, Schwartz BR, Iwata A, W Liles C, Raines EW, Harlan JM. Hematopoietic Fas deficiency does not affect experimental atherosclerotic lesion formation despite inducing a proatherogenic state. Am J Pathol. 2011 ;178(6):2931-7.
Darwish I, Mubareka S, W Liles C. Immunomodulatory therapy for severe influenza. Expert Rev Anti Infect Ther. 2011 ;9(7):807-22.
Higgins SJ, Kain KC, W Liles C. Immunopathogenesis of falciparum malaria: implications for adjunctive therapy in the management of severe and cerebral malaria. Expert Rev Anti Infect Ther. 2011 ;9(9):803-19.
Hawkes M, Opoka RO, Namasopo S, Miller C, Thorpe KE, Lavery JV, Conroy AL, W Liles C, John CC, Kain KC. Inhaled nitric oxide for the adjunctive therapy of severe malaria: protocol for a randomized controlled trial. Trials. 2011 ;12:176.
Serghides L, Kim H, Lu Z, Kain DC, Miller C, Francis RC, W Liles C, Zapol WM, Kain KC. Inhaled nitric oxide reduces endothelial activation and parasite accumulation in the brain, and enhances survival in experimental cerebral malaria. PLoS One. 2011 ;6(11):e27714.
Hawkes M, Opoka ROpika, Namasopo S, Miller C, Conroy AL, Serghides L, Kim H, Thampi N, W Liles C, John CC, et al. Nitric oxide for the adjunctive treatment of severe malaria: hypothesis and rationale. Med Hypotheses. 2011 ;77(3):437-44.
Tsunooka N, Hirayama S, Medin JA, W Liles C, Keshavjee S, Waddell TK. A novel tissue-engineered approach to problems of the postpneumonectomy space. Ann Thorac Surg. 2011 ;91(3):880-6.
Bunce PE, High SM, Nadjafi M, Stanley K, W Liles C, Christian MD. Pandemic H1N1 influenza infection and vascular thrombosis. Clin Infect Dis. 2011 ;52(2):e14-7.
Conroy AL, W Liles C, Molyneux ME, Rogerson SJ, Kain KC. Performance characteristics of combinations of host biomarkers to identify women with occult placental malaria: a case-control study from Malawi. PLoS One. 2011 ;6(12):e28540.
Finney CAm, Hawkes CA, Kain DC, Dhabangi A, Musoke C, Cserti-Gazdewich C, Oravecz T, W Liles C, Kain KC. S1P is associated with protection in human and experimental cerebral malaria. Mol Med. 2011 ;17(7-8):717-25.
Page AV, Kotb M, McGeer A, Low DE, Kain KC, W Liles C. Systemic dysregulation of angiopoietin-1/2 in streptococcal toxic shock syndrome. Clin Infect Dis. 2011 ;52(8):e157-61.
2010
Hawkes M, Li X, Crockett M, Diassiti A, Finney C, Min-Oo G, W Liles C, Liu J, Kain KC. CD36 deficiency attenuates experimental mycobacterial infection. BMC Infect Dis. 2010 ;10:299.
Boggild AK, Nicks BA, Yen L, Van Voorhis W, McMullen R, Buckner FS, W Liles C. Delusional parasitosis: six-year experience with 23 consecutive cases at an academic medical center. Int J Infect Dis. 2010 ;14(4):e317-21.
Finney CAM, Lu Z, Hawkes M, Yeh W-C, W Liles C, Kain KC. Divergent roles of IRAK4-mediated innate immune responses in two experimental models of severe malaria. Am J Trop Med Hyg. 2010 ;83(1):69-74.
Morris AM, Stewart TE, Shandling M, McIntaggart S, W Liles C. Establishing an antimicrobial stewardship program. Healthc Q. 2010 ;13(2):64-70.
Wang F, Lu Z, Hawkes M, Yang H, Kain KC, W Liles C. Fas (CD95) induces rapid, TLR4/IRAK4-dependent release of pro-inflammatory HMGB1 from macrophages. J Inflamm (Lond). 2010 ;7:30.
Hawkes M, Li X, Crockett M, Diassiti A, W Liles C, Liu J, Kain KC. Malaria exacerbates experimental mycobacterial infection in vitro and in vivo. Microbes Infect. 2010 ;12(11):864-74.
Chun CD, W Liles C, Frevert CW, Glenny RW, Altemeier WA. Mechanical ventilation modulates Toll-like receptor-3-induced lung inflammation via a MyD88-dependent, TLR4-independent pathway: a controlled animal study. BMC Pulm Med. 2010 ;10:57.
Mei SHJ, Haitsma JJ, Santos CC dos, Deng Y, Lai PFH, Slutsky AS, W Liles C, Stewart DJ. Mesenchymal stem cells reduce inflammation while enhancing bacterial clearance and improving survival in sepsis. Am J Respir Crit Care Med. 2010 ;182(8):1047-57.

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