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CERID Bibliography
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Author Title Type [ Year] Filters: Keyword is Mice and Author is Kain, Kevin C [Clear All Filters]
ABO blood groups influence macrophage-mediated phagocytosis of Plasmodium falciparum-infected erythrocytes. PLoS Pathog. 2012 ;8(10):e1002942.
. Inhaled nitric oxide therapy fails to improve outcome in experimental severe influenza. Int J Med Sci. 2012 ;9(2):157-62.
. Inhaled nitric oxide reduces endothelial activation and parasite accumulation in the brain, and enhances survival in experimental cerebral malaria. PLoS One. 2011 ;6(11):e27714.
. S1P is associated with protection in human and experimental cerebral malaria. Mol Med. 2011 ;17(7-8):717-25.
. CD36 deficiency attenuates experimental mycobacterial infection. BMC Infect Dis. 2010 ;10:299.
. Divergent roles of IRAK4-mediated innate immune responses in two experimental models of severe malaria. Am J Trop Med Hyg. 2010 ;83(1):69-74.
. Malaria exacerbates experimental mycobacterial infection in vitro and in vivo. Microbes Infect. 2010 ;12(11):864-74.
. Rosiglitazone modulates the innate immune response to Plasmodium falciparum infection and improves outcome in experimental cerebral malaria. J Infect Dis. 2009 ;199(10):1536-45.
. Statins fail to improve outcome in experimental cerebral malaria and potentiate Toll-like receptor-mediated cytokine production by murine macrophages. Am J Trop Med Hyg. 2009 ;81(4):631-7.
. C5 deficiency and C5a or C5aR blockade protects against cerebral malaria. J Exp Med. 2008 ;205(5):1133-43.
Failure of two distinct anti-apoptotic approaches to reduce mortality in experimental cerebral malaria. Am J Trop Med Hyg. 2008 ;79(6):823-5.
. Parasite burden and CD36-mediated sequestration are determinants of acute lung injury in an experimental malaria model. PLoS Pathog. 2008 ;4(5):e1000068.
. Expression microarray analysis implicates apoptosis and interferon-responsive mechanisms in susceptibility to experimental cerebral malaria. Am J Pathol. 2007 ;171(6):1894-903.
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