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CERID Bibliography
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Author Title Type [ Year] Filters: Keyword is Mice and Author is Liles, W Conrad [Clear All Filters]
ABO blood groups influence macrophage-mediated phagocytosis of Plasmodium falciparum-infected erythrocytes. PLoS Pathog. 2012 ;8(10):e1002942.
. The CXCR4/CXCR7/SDF-1 pathway contributes to the pathogenesis of Shiga toxin-associated hemolytic uremic syndrome in humans and mice. J Clin Invest. 2012 ;122(2):759-76.
Inhaled nitric oxide therapy fails to improve outcome in experimental severe influenza. Int J Med Sci. 2012 ;9(2):157-62.
. Network analysis of transcriptional responses induced by mesenchymal stem cell treatment of experimental sepsis. Am J Pathol. 2012 ;181(5):1681-92.
. Hematopoietic Fas deficiency does not affect experimental atherosclerotic lesion formation despite inducing a proatherogenic state. Am J Pathol. 2011 ;178(6):2931-7.
. Inhaled nitric oxide reduces endothelial activation and parasite accumulation in the brain, and enhances survival in experimental cerebral malaria. PLoS One. 2011 ;6(11):e27714.
. A novel tissue-engineered approach to problems of the postpneumonectomy space. Ann Thorac Surg. 2011 ;91(3):880-6.
. S1P is associated with protection in human and experimental cerebral malaria. Mol Med. 2011 ;17(7-8):717-25.
. CD36 deficiency attenuates experimental mycobacterial infection. BMC Infect Dis. 2010 ;10:299.
. Divergent roles of IRAK4-mediated innate immune responses in two experimental models of severe malaria. Am J Trop Med Hyg. 2010 ;83(1):69-74.
. Malaria exacerbates experimental mycobacterial infection in vitro and in vivo. Microbes Infect. 2010 ;12(11):864-74.
. Mechanical ventilation modulates Toll-like receptor-3-induced lung inflammation via a MyD88-dependent, TLR4-independent pathway: a controlled animal study. BMC Pulm Med. 2010 ;10:57.
. Mesenchymal stem cells reduce inflammation while enhancing bacterial clearance and improving survival in sepsis. Am J Respir Crit Care Med. 2010 ;182(8):1047-57.
. Noninjurious mechanical ventilation activates a proinflammatory transcriptional program in the lung. Physiol Genomics. 2009 ;37(3):239-48.
. Rosiglitazone modulates the innate immune response to Plasmodium falciparum infection and improves outcome in experimental cerebral malaria. J Infect Dis. 2009 ;199(10):1536-45.
. Statins fail to improve outcome in experimental cerebral malaria and potentiate Toll-like receptor-mediated cytokine production by murine macrophages. Am J Trop Med Hyg. 2009 ;81(4):631-7.
. C5 deficiency and C5a or C5aR blockade protects against cerebral malaria. J Exp Med. 2008 ;205(5):1133-43.
Failure of two distinct anti-apoptotic approaches to reduce mortality in experimental cerebral malaria. Am J Trop Med Hyg. 2008 ;79(6):823-5.
. Parasite burden and CD36-mediated sequestration are determinants of acute lung injury in an experimental malaria model. PLoS Pathog. 2008 ;4(5):e1000068.
. Expression microarray analysis implicates apoptosis and interferon-responsive mechanisms in susceptibility to experimental cerebral malaria. Am J Pathol. 2007 ;171(6):1894-903.
. Fas (CD95) induces macrophage proinflammatory chemokine production via a MyD88-dependent, caspase-independent pathway. J Leukoc Biol. 2007 ;82(3):721-8.
. Prevention of LPS-induced acute lung injury in mice by mesenchymal stem cells overexpressing angiopoietin 1. PLoS Med. 2007 ;4(9):e269.
. PSGL-1 and mTOR regulate translation of ROCK-1 and physiological functions of macrophages. EMBO J. 2007 ;26(2):505-15.
. Computational identification of key biological modules and transcription factors in acute lung injury. Am J Respir Crit Care Med. 2006 ;173(6):653-8.
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