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CERID Bibliography

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Filters: Keyword is Animals and Author is Liles, W Conrad  [Clear All Filters]
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A
Wolofsky KT, Ayi K, Branch DR, Hult AK, Olsson ML, W Liles C, Cserti-Gazdewich CM, Kain KC. ABO blood groups influence macrophage-mediated phagocytosis of Plasmodium falciparum-infected erythrocytes. PLoS Pathog. 2012 ;8(10):e1002942.
Ayi K, W Liles C, Gros P, Kain KC. Adenosine triphosphate depletion of erythrocytes simulates the phenotype associated with pyruvate kinase deficiency and confers protection against Plasmodium falciparum in vitro. J Infect Dis. 2009 ;200(8):1289-99.
W Liles C. Is angiopoietin-2 the key piece in the sepsis puzzle?*. Crit Care Med. 2012 ;40(11):3100-1.
B
Matute-Bello G, W Liles C, Frevert CW, Dhanireddy S, Ballman K, Wong V, Green RR, Song HYeong, Witcher DR, Jakubowski JA, et al. Blockade of the Fas/FasL system improves pneumococcal clearance from the lungs without preventing dissemination of bacteria to the spleen. J Infect Dis. 2005 ;191(4):596-606.
C
Patel SN, Berghout J, Lovegrove FE, Ayi K, Conroy A, Serghides L, Min-Oo G, D Gowda C, J Sarma V, Rittirsch D, et al. C5 deficiency and C5a or C5aR blockade protects against cerebral malaria. J Exp Med. 2008 ;205(5):1133-43.
Conroy A, Serghides L, Finney C, Owino SO, Kumar S, D Gowda C, W Liles C, Moore JM, Kain KC. C5a enhances dysregulated inflammatory and angiogenic responses to malaria in vitro: potential implications for placental malaria. PLoS One. 2009 ;4(3):e4953.
Hawkes M, Li X, Crockett M, Diassiti A, Finney C, Min-Oo G, W Liles C, Liu J, Kain KC. CD36 deficiency attenuates experimental mycobacterial infection. BMC Infect Dis. 2010 ;10:299.
Yaraei K, Campbell LAnn, Zhu X, W Liles C, Kuo C-C, Rosenfeld ME. Chlamydia pneumoniae augments the oxidized low-density lipoprotein-induced death of mouse macrophages by a caspase-independent pathway. Infect Immun. 2005 ;73(7):4315-22.
Gharib SA, W Liles C, Matute-Bello G, Glenny RW, Martin TR, Altemeier WA. Computational identification of key biological modules and transcription factors in acute lung injury. Am J Respir Crit Care Med. 2006 ;173(6):653-8.
Petruzziello-Pellegrini TN, Yuen DA, Page AV, Patel S, Soltyk AM, Matouk CC, Wong DK, Turgeon PJ, Fish JE, Ho JJDavid, et al. The CXCR4/CXCR7/SDF-1 pathway contributes to the pathogenesis of Shiga toxin-associated hemolytic uremic syndrome in humans and mice. J Clin Invest. 2012 ;122(2):759-76.
D
Boggild AK, Page AV, Keystone JS, Morris AM, W Liles C. Delay in diagnosis: malaria in a returning traveller. CMAJ. 2009 ;180(11):1129-31.
Marr KA, S Balajee A, Hawn TR, Ozinsky A, Pham U, Akira S, Aderem A, W Liles C. Differential role of MyD88 in macrophage-mediated responses to opportunistic fungal pathogens. Infect Immun. 2003 ;71(9):5280-6.
Finney CAM, Lu Z, Hawkes M, Yeh W-C, W Liles C, Kain KC. Divergent roles of IRAK4-mediated innate immune responses in two experimental models of severe malaria. Am J Trop Med Hyg. 2010 ;83(1):69-74.
E
Yaraei K, Campbell LAnn, Zhu X, W Liles C, Kuo C-C, Rosenfeld ME. Effect of Chlamydia pneumoniae on cellular ATP content in mouse macrophages: role of Toll-like receptor 2. Infect Immun. 2005 ;73(7):4323-6.
Kim H, Higgins S, W Liles C, Kain KC. Endothelial activation and dysregulation in malaria: a potential target for novel therapeutics. Curr Opin Hematol. 2011 ;18(3):177-85.
Lee WL, W Liles C. Endothelial activation, dysfunction and permeability during severe infections. Curr Opin Hematol. 2011 ;18(3):191-6.
Lovegrove FE, Gharib SA, Patel SN, Hawkes CA, Kain KC, W Liles C. Expression microarray analysis implicates apoptosis and interferon-responsive mechanisms in susceptibility to experimental cerebral malaria. Am J Pathol. 2007 ;171(6):1894-903.
F
Helmers AJ, Lovegrove FE, Harlan JM, Kain KC, W Liles C. Failure of two distinct anti-apoptotic approaches to reduce mortality in experimental cerebral malaria. Am J Trop Med Hyg. 2008 ;79(6):823-5.
Altemeier WA, Zhu X, Berrington WR, Harlan JM, W Liles C. Fas (CD95) induces macrophage proinflammatory chemokine production via a MyD88-dependent, caspase-independent pathway. J Leukoc Biol. 2007 ;82(3):721-8.
Matute-Bello G, Lee JS, W Liles C, Frevert CW, Mongovin S, Wong V, Ballman K, Sutlief S, Martin TR. Fas-mediated acute lung injury requires fas expression on nonmyeloid cells of the lung. J Immunol. 2005 ;175(6):4069-75.
G
Page AV, W Liles C. Granulocyte colony-stimulating factor, granulocyte-macrophage colony-stimulating factor, and other immunomodulatory therapies for the treatment of infectious diseases in solid organ transplant recipients. Curr Opin Organ Transplant. 2008 ;13(6):575-80.
H
R de Claro A, Zhu X, Tang J, Morgan-Stevenson V, Schwartz BR, Iwata A, W Liles C, Raines EW, Harlan JM. Hematopoietic Fas deficiency does not affect experimental atherosclerotic lesion formation despite inducing a proatherogenic state. Am J Pathol. 2011 ;178(6):2931-7.
I
Serghides L, Kim H, Lu Z, Kain DC, Miller C, Francis RC, W Liles C, Zapol WM, Kain KC. Inhaled nitric oxide reduces endothelial activation and parasite accumulation in the brain, and enhances survival in experimental cerebral malaria. PLoS One. 2011 ;6(11):e27714.
Darwish I, Miller C, Kain KC, W Liles C. Inhaled nitric oxide therapy fails to improve outcome in experimental severe influenza. Int J Med Sci. 2012 ;9(2):157-62.
L
Kaushansky K, Fox N, Lin NL, W Liles C. Lineage-specific growth factors can compensate for stem and progenitor cell deficiencies at the postprogenitor cell level: an analysis of doubly TPO- and G-CSF receptor-deficient mice. Blood. 2002 ;99(10):3573-8.

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